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Inosine

User Ratings for Immune System Support treatments
Out of 10
3.7  Effectiveness
2.3  Side Effects
3.5  Holistic Benefits

Be better informed. Are you taking Inosine?  
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Treatment Information
This is a chemical and can be prepared in a laboratory setting. Used in manufacturing medicine.

It is known by other names like Hypoxanthine Riboside, Hypoxanthosine, 2,3-Diphosphoglycerate, 6-9 Dihydro-9-B-D-ribofuranosyl-1H-puin-6-one, 9-B-D-ribofuranosylhypoxanthine.

Proposed use includes treating various types of cardiovascular diseases.

Dosage of this medication is mostly customized and varies from one individual to another since it depends on various factors like age and prevailing health of the person.
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About Inosine
This is a chemical and can be prepared in a laboratory setting. Used in manufacturing medicine. It is known by other names like Hypoxanthine Riboside, Hypoxanthosine, 2,3-Diphosphoglycerate, 6-9 Dihydro-9-B-D-ribofuranosyl-1H-puin-6-one, 9-B-D-ribofuranosylhypoxanthine. Proposed use includes treating various types of cardiovascular diseases. Dosage of this medication is mostly customized and varies from one individual to another since it depends on various factors like age and prevailing health of the person.
About Inosine
Inosine is a nucleoside that is formed when hypoxanthine is attached to a ribose ring (also known as a ribofuranose) via a β-N9-glycosidic bond. Inosine is commonly found in tRNAs and is essential for proper translation of the genetic code in wobble base pairs. Knowledge of inosine metabolism has led to advances in immunotherapy in recent decades. Inosine monophosphate is oxidised by the enzyme inosine monophosphate dehydrogenase yielding xanthosine monophosphate, a key precursor in purine metabolism. Mycophenolate mofetil is an anti-metabolite, anti-proliferative drug, used in the treatment of a variety of autoimmune diseases including Wegener's granulomatosis. The uptake of purine by actively dividing B cells can exceed 8 times that of normal body cells and therefore this set of white cells (which cannot operate purine salvage pathways) is selectively targeted by the purine deficiency resulting from IMD inhibition.

Source: Wikipedia




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